ATW2015 INCLUDES SESSIONS ON NEUROGENERATION AND NON-TRADITIONAL ROLES OF THE A-T PROTEIN

Scientists and clinicians from around the world traveled to Beijing, China in October to share information at the 2015 A-T Workshop, a meeting co-sponsored by the A-T Children’s Project and held at the Capital Normal University.

Beyond primarily discussing DNA repair roles of the ATM protein as in the past, this year’s meeting included sessions on: 1) other stresses that activate ATM without damaging DNA; 2) how the loss of the ATM protein may lead to sick and dying brain cells; and, 3) clinical aspects of A-T. Presentations by clinicians, established and young investigators, as well as special keynote lectures, including the A-T Children’s Project Keynote presented by Michael Kastan, MD, PhD, prompted provocative discussions.

Importantly, the meeting provided opportunities for scientists who spend most of their time in laboratories to interact with clinicians who treat patients with A-T, exposing gaps in their knowledge about this disease and promoting discussions on how best to address them.

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